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Disinhibition is often accompanied by other coinciding effects such as [[amnesia]] and [[anxiety suppression]] in a manner which can further decrease the person's observance of and regard for social norms. It is most commonly induced under the influence of [[dosage#common|moderate]] [[dosage|dosages]] of [[GABAergic]] [[depressant|depressants]], such as [[alcohol]],<ref name="BettingerTopper2014">{{cite journal|last1=Bettinger|first1=Jill C.|last2=Topper|first2=Stephen M.|last3=Aguilar|first3=Sara C.|last4=Topper|first4=Viktoria Y.|last5=Elbel|first5=Erin|last6=Pierce-Shimomura|first6=Jonathan T.|title=Alcohol Disinhibition of Behaviors in C. elegans|journal=PLoS ONE|volume=9|issue=3|year=2014|pages=e92965|issn=1932-6203|doi=10.1371/journal.pone.0092965}}</ref> [[benzodiazepine|benzodiazepines]],<ref name="Paton2018">{{cite journal|last1=Paton|first1=Carol|title=Benzodiazepines and disinhibition: a review|journal=Psychiatric Bulletin|volume=26|issue=12|year=2018|pages=460–462|issn=0955-6036|doi=10.1192/pb.26.12.460}}</ref> [[phenibut]], and [[GHB]]. However, it may also occur under the influence of certain [[stimulant|stimulants]],<ref name="Fillmore2003">{{cite journal|last1=Fillmore|first1=M|title=Effects of d-amphetamine on behavioral control in stimulant abusers: the role of prepotent response tendencies|journal=Drug and Alcohol Dependence|volume=71|issue=2|year=2003|pages=143–152|issn=03768716|doi=10.1016/S0376-8716(03)00089-9}}</ref> [[entactogen|entactogens]],<ref name="AndoBenko2006">{{cite journal|last1=Ando|first1=Romeo D.|last2=Benko|first2=Anita|last3=Ferrington|first3=Linda|last4=Kirilly|first4=Eszter|last5=Kelly|first5=Paul A.T.|last6=Bagdy|first6=Gyorgy|title=Partial lesion of the serotonergic system by a single dose of MDMA results in behavioural disinhibition and enhances acute MDMA-induced social behaviour on the social interaction test|journal=Neuropharmacology|volume=50|issue=7|year=2006|pages=884–896|issn=00283908|doi=10.1016/j.neuropharm.2005.12.010}}</ref> and [[dissociative|dissociatives]]<ref name="LissekGüntürkün2003">{{cite journal|last1=Lissek|first1=Silke|last2=Güntürkün|first2=Onur|title=Dissociation of Extinction and Behavioral Disinhibition: The Role of NMDA Receptors in the Pigeon Associative Forebrain during Extinction|journal=The Journal of Neuroscience|volume=23|issue=22|year=2003|pages=8119–8124|issn=0270-6474|doi=10.1523/JNEUROSCI.23-22-08119.2003}}</ref>.
Disinhibition is often accompanied by other coinciding effects such as [[amnesia]] and [[anxiety suppression]] in a manner which can further decrease the person's observance of and regard for social norms. It is most commonly induced under the influence of [[dosage#common|moderate]] [[dosage|dosages]] of [[GABAergic]] [[depressant|depressants]], such as [[alcohol]],<ref name="BettingerTopper2014">{{cite journal|last1=Bettinger|first1=Jill C.|last2=Topper|first2=Stephen M.|last3=Aguilar|first3=Sara C.|last4=Topper|first4=Viktoria Y.|last5=Elbel|first5=Erin|last6=Pierce-Shimomura|first6=Jonathan T.|title=Alcohol Disinhibition of Behaviors in C. elegans|journal=PLoS ONE|volume=9|issue=3|year=2014|pages=e92965|issn=1932-6203|doi=10.1371/journal.pone.0092965}}</ref> [[benzodiazepine|benzodiazepines]],<ref name="Paton2018">{{cite journal|last1=Paton|first1=Carol|title=Benzodiazepines and disinhibition: a review|journal=Psychiatric Bulletin|volume=26|issue=12|year=2018|pages=460–462|issn=0955-6036|doi=10.1192/pb.26.12.460}}</ref> [[phenibut]], and [[GHB]]. However, it may also occur under the influence of certain [[stimulant|stimulants]],<ref name="Fillmore2003">{{cite journal|last1=Fillmore|first1=M|title=Effects of d-amphetamine on behavioral control in stimulant abusers: the role of prepotent response tendencies|journal=Drug and Alcohol Dependence|volume=71|issue=2|year=2003|pages=143–152|issn=03768716|doi=10.1016/S0376-8716(03)00089-9}}</ref> [[entactogen|entactogens]],<ref name="AndoBenko2006">{{cite journal|last1=Ando|first1=Romeo D.|last2=Benko|first2=Anita|last3=Ferrington|first3=Linda|last4=Kirilly|first4=Eszter|last5=Kelly|first5=Paul A.T.|last6=Bagdy|first6=Gyorgy|title=Partial lesion of the serotonergic system by a single dose of MDMA results in behavioural disinhibition and enhances acute MDMA-induced social behaviour on the social interaction test|journal=Neuropharmacology|volume=50|issue=7|year=2006|pages=884–896|issn=00283908|doi=10.1016/j.neuropharm.2005.12.010}}</ref> and [[dissociative|dissociatives]]<ref name="LissekGüntürkün2003">{{cite journal|last1=Lissek|first1=Silke|last2=Güntürkün|first2=Onur|title=Dissociation of Extinction and Behavioral Disinhibition: The Role of NMDA Receptors in the Pigeon Associative Forebrain during Extinction|journal=The Journal of Neuroscience|volume=23|issue=22|year=2003|pages=8119–8124|issn=0270-6474|doi=10.1523/JNEUROSCI.23-22-08119.2003}}</ref>.
Above is a perspective from psychology and limited to mind, which is only one of three aspects of what we are. From a soul perspective, inhibited is meaning untrue, not true to self, hiding, pretention of who one is. This ranges from on the low end to highlighting certain aspects of self and minimizing others less desirable traits to on the high end out right entirely fabricated 'personality' where one believes that essentially no aspect of themselves is acceptable or loveable as is. "Unable to act in a relaxed and natural way because of self-consciousness or mental restraint". The Ego is the source of all "mental restraint", or lying, pretending, mistruth, acting.  All distortion no matter how small, of self, of truth in a moment, is absolute self denial. There are no degrees of truth. One is either true to the moment and to self or one is a ego or mental construction that one made to feel "safe" from other egos. So from souls perspective, to be un inhibited is to be the truth of yourself. When we are the truth with each other, we have experiences of love because in this moment we finally get to meet each other vs seeing only masks. Love is free as a innate quality, as such it can not be present in the environment of a lie no matter how small. It is why our day to day experiences are so loveless and why we crave the feeling and the freeness of substances that give us a temporary permission to be free, to be what we really are in a moment. The future evolution of Humanity will see these substances become less and less relevant as we evolve back to a natural state vs a 'normal' or 'average' state of self delusion.
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===Psychoactive substances===
===Psychoactive substances===
Compounds within our [[psychoactive substance index]] which may cause this effect include:
Compounds within our [[psychoactive substance index]] which may cause this effect include:

Revision as of 20:26, 10 May 2022

Disinhibition is medically recognized as an orientation towards immediate gratification, leading to impulsive behavior driven by current thoughts, feelings, and external stimuli, without regard for past learning or consideration of future consequences.[1][2][3] This is usually manifested through recklessness, poor risk assessment, and a disregard for social conventions.

At its lower levels of intensity, disinhibition can allow one to overcome emotional apprehension and suppressed social skills in a manner that is moderated and controllable for the average person. This can often prove useful for those who suffer from social anxiety or a general lack of self-confidence.

However, at higher levels of intensity, the disinhibited individual may be completely unable to maintain any semblance of self-restraint, at the expense of politeness, sensitivity, social appropriateness, or local laws and regulations. This lack of constraint can be negative, neutral, or positive depending on the individual and their current environment. The negative consequences of disinhibited behaviour range from relatively benign consequences (such as embarrassing oneself) to destructive and damaging ones (such as driving under the influence or committing criminal acts).

Disinhibition is often accompanied by other coinciding effects such as amnesia and anxiety suppression in a manner which can further decrease the person's observance of and regard for social norms. It is most commonly induced under the influence of moderate dosages of GABAergic depressants, such as alcohol,[4] benzodiazepines,[5] phenibut, and GHB. However, it may also occur under the influence of certain stimulants,[6] entactogens,[7] and dissociatives[8].


Above is a perspective from psychology and limited to mind, which is only one of three aspects of what we are. From a soul perspective, inhibited is meaning untrue, not true to self, hiding, pretention of who one is. This ranges from on the low end to highlighting certain aspects of self and minimizing others less desirable traits to on the high end out right entirely fabricated 'personality' where one believes that essentially no aspect of themselves is acceptable or loveable as is. "Unable to act in a relaxed and natural way because of self-consciousness or mental restraint". The Ego is the source of all "mental restraint", or lying, pretending, mistruth, acting. All distortion no matter how small, of self, of truth in a moment, is absolute self denial. There are no degrees of truth. One is either true to the moment and to self or one is a ego or mental construction that one made to feel "safe" from other egos. So from souls perspective, to be un inhibited is to be the truth of yourself. When we are the truth with each other, we have experiences of love because in this moment we finally get to meet each other vs seeing only masks. Love is free as a innate quality, as such it can not be present in the environment of a lie no matter how small. It is why our day to day experiences are so loveless and why we crave the feeling and the freeness of substances that give us a temporary permission to be free, to be what we really are in a moment. The future evolution of Humanity will see these substances become less and less relevant as we evolve back to a natural state vs a 'normal' or 'average' state of self delusion.


Psychoactive substances

Compounds within our psychoactive substance index which may cause this effect include:

... further results

Experience reports

Annectdotal reports which describe this effect with our experience index include:

See also

References

  1. ↑ "Glossary of Technical Terms". Diagnostic and statistical manual of mental disorders (5th ed.): 820. 2013. doi:10.1176/appi.books.9780890425596.GlossaryofTechnicalTerms. 
  2. ↑ Zamboni, G.; Huey, E. D.; Krueger, F.; Nichelli, P. F.; Grafman, J. (2008). "Apathy and disinhibition in frontotemporal dementia: Insights into their neural correlates". Neurology. 71 (10): 736–742. doi:10.1212/01.wnl.0000324920.96835.95. ISSN 0028-3878. 
  3. ↑ Källmén, Håkan; Gustafson, Roland (1998). "Alcohol and Disinhibition". European Addiction Research. 4 (4): 150–162. doi:10.1159/000018948. ISSN 1022-6877. 
  4. ↑ Bettinger, Jill C.; Topper, Stephen M.; Aguilar, Sara C.; Topper, Viktoria Y.; Elbel, Erin; Pierce-Shimomura, Jonathan T. (2014). "Alcohol Disinhibition of Behaviors in C. elegans". PLoS ONE. 9 (3): e92965. doi:10.1371/journal.pone.0092965. ISSN 1932-6203. 
  5. ↑ Paton, Carol (2018). "Benzodiazepines and disinhibition: a review". Psychiatric Bulletin. 26 (12): 460–462. doi:10.1192/pb.26.12.460. ISSN 0955-6036. 
  6. ↑ Fillmore, M (2003). "Effects of d-amphetamine on behavioral control in stimulant abusers: the role of prepotent response tendencies". Drug and Alcohol Dependence. 71 (2): 143–152. doi:10.1016/S0376-8716(03)00089-9. ISSN 0376-8716. 
  7. ↑ Ando, Romeo D.; Benko, Anita; Ferrington, Linda; Kirilly, Eszter; Kelly, Paul A.T.; Bagdy, Gyorgy (2006). "Partial lesion of the serotonergic system by a single dose of MDMA results in behavioural disinhibition and enhances acute MDMA-induced social behaviour on the social interaction test". Neuropharmacology. 50 (7): 884–896. doi:10.1016/j.neuropharm.2005.12.010. ISSN 0028-3908. 
  8. ↑ Lissek, Silke; Güntürkün, Onur (2003). "Dissociation of Extinction and Behavioral Disinhibition: The Role of NMDA Receptors in the Pigeon Associative Forebrain during Extinction". The Journal of Neuroscience. 23 (22): 8119–8124. doi:10.1523/JNEUROSCI.23-22-08119.2003. ISSN 0270-6474. 
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