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'''Dream suppression''' is a decrease in the vividness, intensity, frequency, and recollection of a person's dreams. At its lower levels, this can be a partial suppression which results in the person having dreams of a lesser intensity and a lower rate of frequency. However, at its higher levels, this can be a complete suppression which results in the person not experiencing any dreams at all.
'''Dream suppression''' is a decrease in the vividness, intensity, frequency, and recollection of a person's dreams. At its lower levels, this can be a partial suppression which results in the person having dreams of a lesser intensity and a lower rate of frequency. However, at its higher levels, this can be a complete suppression which results in the person not experiencing any dreams at all.


Dream suppression is most commonly induced under the influence of [[dosage#common|moderate]] [[dosage|dosages]] of [[cannabinoid|cannabinoids]]<ref name="Schierenbeck2008">Schierenbeck, T., Riemann, D., Berger, M., & Hornyak, M. (2008). Effect of illicit recreational drugs upon sleep: cocaine, ecstasy and marijuana. Sleep medicine reviews, 12(5), 381-389. https://doi.org/10.1016/j.smrv.2007.12.004</ref> and most types of antidepressants<ref name="Sharpley1995">Sharpley, A. L., & Cowen, P. J. (1995). Effect of pharmacologic treatments on the sleep of depressed patients. Biological psychiatry, 37(2), 85-98. https://doi.org/10.1016/0006-3223(94)00135-P</ref><ref name="Trivedi1999">Trivedi, M. H., Rush, A. J., Armitage, R., Gullion, C. M., Grannemann, B. D., Orsulak, P. J., & Roffwarg, H. P. (1999). Effects of fluoxetine on the polysomnogram in outpatients with major depression. Neuropsychopharmacology, 20(5), 447-459. https://doi.org/10.1016/S0893-133X(98)00131-6</ref><ref>Vogel, G. W., Buffenstein, A., Minter, K., & Hennessey, A. (1990). Drug effects on REM sleep and on endogenous depression. Neuroscience & Biobehavioral Reviews, 14(1), 49-63.
Dream suppression is most commonly induced under the influence of [[dosage#common|moderate]] [[dosage|dosages]] of [[cannabinoid|cannabinoids]]<ref name="SchierenbeckRiemann2008">{{cite journal|last1=Schierenbeck|first1=Thomas|last2=Riemann|first2=Dieter|last3=Berger|first3=Mathias|last4=Hornyak|first4=Magdolna|title=Effect of illicit recreational drugs upon sleep: Cocaine, ecstasy and marijuana|journal=Sleep Medicine Reviews|volume=12|issue=5|year=2008|pages=381–389|issn=10870792|doi=10.1016/j.smrv.2007.12.004}}</ref> and most types of antidepressants<ref name="SharpleyCowen1995">{{cite journal|last1=Sharpley|first1=Ann L.|last2=Cowen|first2=Philip J.|title=Effect of pharmacologic treatments on the sleep of depressed patients|journal=Biological Psychiatry|volume=37|issue=2|year=1995|pages=85–98|issn=00063223|doi=10.1016/0006-3223(94)00135-P}}</ref><ref name="Trivedi1999">{{cite journal|last1=Trivedi|first1=M|title=Effects of Fluoxetine on the Polysomnogram in Outpatients with Major Depression|journal=Neuropsychopharmacology|volume=20|issue=5|year=1999|pages=447–459|issn=0893133X|doi=10.1016/S0893-133X(98)00131-6}}</ref><ref name="VogelBuffenstein1990">{{cite journal|last1=Vogel|first1=G.W.|last2=Buffenstein|first2=A.|last3=Minter|first3=K.|last4=Hennessey|first4=Ann|title=Drug effects on REM sleep and on endogenous depression|journal=Neuroscience & Biobehavioral Reviews|volume=14|issue=1|year=1990|pages=49–63|issn=01497634|doi=10.1016/S0149-7634(05)80159-9}}</ref>. This is due to the way in which they increase REM latency, decrease REM sleep, reduce total sleep time and efficiency, and increase [[wakefulness]].<ref name="SchierenbeckRiemann2008"/><ref name="SharpleyCowen1995"/><ref name="Trivedi1999"/><ref>Feinberg, I., Jones, R., Walker, J. M., Cavness, C., & March, J. (1975). Effects of high dosage delta‐9‐tetrahydrocannabinol on sleep patterns in man. Clinical Pharmacology & Therapeutics, 17(4), 458-466. https://www.ncbi.nlm.nih.gov/pubmed/164314</ref> REM sleep is where the majority of dreams occur.<ref>Hobson, J. A., Stickgold, R., & Pace-Schott, E. F. (1998). The neuropsychology of REM sleep dreaming. Neuroreport, 9(3), R1-R14. https://www.ncbi.nlm.nih.gov/pubmed/9512371</ref>
https://doi.org/10.1016/S0149-7634(05)80159-9</ref>. This is due to the way in which they increase REM latency, decrease REM sleep, reduce total sleep time and efficiency, and increase [[wakefulness]].<ref name="Schierenbeck2008"/><ref name="Sharpley1995"/><ref name="Trivedi1999"/><ref>Feinberg, I., Jones, R., Walker, J. M., Cavness, C., & March, J. (1975). Effects of high dosage delta‐9‐tetrahydrocannabinol on sleep patterns in man. Clinical Pharmacology & Therapeutics, 17(4), 458-466. https://www.ncbi.nlm.nih.gov/pubmed/164314</ref> REM sleep is where the majority of dreams occur.<ref>Hobson, J. A., Stickgold, R., & Pace-Schott, E. F. (1998). The neuropsychology of REM sleep dreaming. Neuroreport, 9(3), R1-R14. https://www.ncbi.nlm.nih.gov/pubmed/9512371</ref>
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===Psychoactive substances===
===Psychoactive substances===

Revision as of 00:23, 8 September 2018

Dream suppression is a decrease in the vividness, intensity, frequency, and recollection of a person's dreams. At its lower levels, this can be a partial suppression which results in the person having dreams of a lesser intensity and a lower rate of frequency. However, at its higher levels, this can be a complete suppression which results in the person not experiencing any dreams at all.

Dream suppression is most commonly induced under the influence of moderate dosages of cannabinoids[1] and most types of antidepressants[2][3][4]. This is due to the way in which they increase REM latency, decrease REM sleep, reduce total sleep time and efficiency, and increase wakefulness.[1][2][3][5] REM sleep is where the majority of dreams occur.[6]

Psychoactive substances

Compounds within our psychoactive substance index which may cause this effect include:

See also

References

  1. ↑ 1.0 1.1 Schierenbeck, Thomas; Riemann, Dieter; Berger, Mathias; Hornyak, Magdolna (2008). "Effect of illicit recreational drugs upon sleep: Cocaine, ecstasy and marijuana". Sleep Medicine Reviews. 12 (5): 381–389. doi:10.1016/j.smrv.2007.12.004. ISSN 1087-0792. 
  2. ↑ 2.0 2.1 Sharpley, Ann L.; Cowen, Philip J. (1995). "Effect of pharmacologic treatments on the sleep of depressed patients". Biological Psychiatry. 37 (2): 85–98. doi:10.1016/0006-3223(94)00135-P. ISSN 0006-3223. 
  3. ↑ 3.0 3.1 Trivedi, M (1999). "Effects of Fluoxetine on the Polysomnogram in Outpatients with Major Depression". Neuropsychopharmacology. 20 (5): 447–459. doi:10.1016/S0893-133X(98)00131-6. ISSN 0893-133X. 
  4. ↑ Vogel, G.W.; Buffenstein, A.; Minter, K.; Hennessey, Ann (1990). "Drug effects on REM sleep and on endogenous depression". Neuroscience & Biobehavioral Reviews. 14 (1): 49–63. doi:10.1016/S0149-7634(05)80159-9. ISSN 0149-7634. 
  5. ↑ Feinberg, I., Jones, R., Walker, J. M., Cavness, C., & March, J. (1975). Effects of high dosage delta‐9‐tetrahydrocannabinol on sleep patterns in man. Clinical Pharmacology & Therapeutics, 17(4), 458-466. https://www.ncbi.nlm.nih.gov/pubmed/164314
  6. ↑ Hobson, J. A., Stickgold, R., & Pace-Schott, E. F. (1998). The neuropsychology of REM sleep dreaming. Neuroreport, 9(3), R1-R14. https://www.ncbi.nlm.nih.gov/pubmed/9512371
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