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[[category:Cognitive suppression]]
<onlyinclude>
'''Suppression of language''' can be described as a cognitive component which partially to entirely interferes with the REM dream stage in the dream cycle or suppresses recollection and retention of the dream in short term memory.  
'''Dream suppression''' is defined as a decrease in the vividness, intensity, frequency, and recollection of a person's dreams. At its lower levels, this can be a partial suppression which results in the person having dreams of a lesser intensity and a lower rate of frequency. However, at its higher levels, this can be a complete suppression which results in the person not experiencing any dreams at all.


This can decrease the percieved frequency and vividness of an REM dream state, leading into a complete lack of ability to recall any details about the dream. This can be the result from one of two different effects, directly effecting the REM dream cycle and indirectly by partially or completely eliminating short term recall of a dream. The first of which can result from use of cannabinoids such as [[THC]] before sleep and the latter from [[depressant]]s such as [[alprazolam]].
Dream suppression is most commonly induced under the influence of [[dosage#common|moderate]] [[dosage|dosages]] of [[cannabinoid|cannabinoids]]<ref name="SchierenbeckRiemann2008">{{cite journal|last1=Schierenbeck|first1=Thomas|last2=Riemann|first2=Dieter|last3=Berger|first3=Mathias|last4=Hornyak|first4=Magdolna|title=Effect of illicit recreational drugs upon sleep: Cocaine, ecstasy and marijuana|journal=Sleep Medicine Reviews|volume=12|issue=5|year=2008|pages=381–389|issn=10870792|doi=10.1016/j.smrv.2007.12.004}}</ref> and most types of antidepressants<ref name="SharpleyCowen1995">{{cite journal|last1=Sharpley|first1=Ann L.|last2=Cowen|first2=Philip J.|title=Effect of pharmacologic treatments on the sleep of depressed patients|journal=Biological Psychiatry|volume=37|issue=2|year=1995|pages=85–98|issn=00063223|doi=10.1016/0006-3223(94)00135-P}}</ref><ref name="Trivedi1999">{{cite journal|last1=Trivedi|first1=M|title=Effects of Fluoxetine on the Polysomnogram in Outpatients with Major Depression|journal=Neuropsychopharmacology|volume=20|issue=5|year=1999|pages=447–459|issn=0893133X|doi=10.1016/S0893-133X(98)00131-6}}</ref><ref name="VogelBuffenstein1990">{{cite journal|last1=Vogel|first1=G.W.|last2=Buffenstein|first2=A.|last3=Minter|first3=K.|last4=Hennessey|first4=Ann|title=Drug effects on REM sleep and on endogenous depression|journal=Neuroscience & Biobehavioral Reviews|volume=14|issue=1|year=1990|pages=49–63|issn=01497634|doi=10.1016/S0149-7634(05)80159-9}}</ref>. This is due to the way in which they increase REM latency, decrease REM sleep, reduce total sleep time and efficiency, and increase [[wakefulness]].<ref name="SchierenbeckRiemann2008" /><ref name="SharpleyCowen1995" /><ref name="Trivedi1999" /><ref>{{cite journal | vauthors=((Feinberg, I.)), ((Jones, R.)), ((Walker, J. M.)), ((Cavness, C.)), ((March, J.)) | journal=Clinical Pharmacology & Therapeutics | title=Effects of high dosage delta-9-tetrahydrocannabinol on sleep patterns in man | volume=17 | issue=4 | pages=458–466 | date= April 1975 | url=https://onlinelibrary.wiley.com/doi/10.1002/cpt1975174458 | issn=00099236 | doi=10.1002/cpt1975174458 | access-date=4 June 2022}}</ref> REM sleep is where the majority of dreams occur.<ref>{{cite journal | vauthors=((Hobson, J. A.)), ((Stickgold, R.)), ((Pace-Schott, E. F.)) | journal=NeuroReport | title=The neuropsychology of REM sleep dreaming: | volume=9 | issue=3 | pages=R1–R14 | date= February 1998 | url=http://journals.lww.com/00001756-199802160-00033 | issn=0959-4965 | doi=10.1097/00001756-199802160-00033 | access-date=4 June 2022}}</ref>
</onlyinclude>
===Psychoactive substances===
Compounds within our [[psychoactive substance index]] which may cause this effect include:
{{#ask:[[Category:Psychoactive substance]][[Effect::Dream suppression]]|format=ul|Columns=2}}
===Experience reports===
Annectdotal reports which describe this effect with our [[experience index]] include:
{{#ask:[[Category:Experience]][[Effect::Dream suppression]]|format=ul|Columns=2}}
===See also===
*[[Responsible use]]
*[[Subjective effects index]]
*[[Dream potentiation]]
*[[Psychedelics#Subjective_effects|Psychedelics - Subjective effects]]
*[[Dissociatives#Subjective_effects|Dissociatives - Subjective effects]]
*[[Deliriants#Subjective_effects|Deliriants - Subjective effects]]
===External links===
*[https://en.wikipedia.org/wiki/Dream Dream (Wikipedia)]
===References===
<references />
[[Category:Cognitive]]
[[Category:Suppression]]
[[Category:Effect]]

Latest revision as of 00:35, 4 June 2022

Dream suppression is defined as a decrease in the vividness, intensity, frequency, and recollection of a person's dreams. At its lower levels, this can be a partial suppression which results in the person having dreams of a lesser intensity and a lower rate of frequency. However, at its higher levels, this can be a complete suppression which results in the person not experiencing any dreams at all.

Dream suppression is most commonly induced under the influence of moderate dosages of cannabinoids[1] and most types of antidepressants[2][3][4]. This is due to the way in which they increase REM latency, decrease REM sleep, reduce total sleep time and efficiency, and increase wakefulness.[1][2][3][5] REM sleep is where the majority of dreams occur.[6]

Psychoactive substances

Compounds within our psychoactive substance index which may cause this effect include:

Experience reports

Annectdotal reports which describe this effect with our experience index include:

See also

References

  1. ↑ 1.0 1.1 Schierenbeck, Thomas; Riemann, Dieter; Berger, Mathias; Hornyak, Magdolna (2008). "Effect of illicit recreational drugs upon sleep: Cocaine, ecstasy and marijuana". Sleep Medicine Reviews. 12 (5): 381–389. doi:10.1016/j.smrv.2007.12.004. ISSN 1087-0792. 
  2. ↑ 2.0 2.1 Sharpley, Ann L.; Cowen, Philip J. (1995). "Effect of pharmacologic treatments on the sleep of depressed patients". Biological Psychiatry. 37 (2): 85–98. doi:10.1016/0006-3223(94)00135-P. ISSN 0006-3223. 
  3. ↑ 3.0 3.1 Trivedi, M (1999). "Effects of Fluoxetine on the Polysomnogram in Outpatients with Major Depression". Neuropsychopharmacology. 20 (5): 447–459. doi:10.1016/S0893-133X(98)00131-6. ISSN 0893-133X. 
  4. ↑ Vogel, G.W.; Buffenstein, A.; Minter, K.; Hennessey, Ann (1990). "Drug effects on REM sleep and on endogenous depression". Neuroscience & Biobehavioral Reviews. 14 (1): 49–63. doi:10.1016/S0149-7634(05)80159-9. ISSN 0149-7634. 
  5. ↑ Feinberg, I., Jones, R., Walker, J. M., Cavness, C., March, J. (April 1975). "Effects of high dosage delta-9-tetrahydrocannabinol on sleep patterns in man". Clinical Pharmacology & Therapeutics. 17 (4): 458–466. doi:10.1002/cpt1975174458. ISSN 0009-9236. Retrieved 4 June 2022. 
  6. ↑ Hobson, J. A., Stickgold, R., Pace-Schott, E. F. (February 1998). "The neuropsychology of REM sleep dreaming:". NeuroReport. 9 (3): R1–R14. doi:10.1097/00001756-199802160-00033. ISSN 0959-4965. Retrieved 4 June 2022. 
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